MRSA
How does MRSA become so severe?
How does MRSA lead to other serious infections?
Is there any special genetic or biological difference that makes the bacteria potent?
How come MRSA is common in hospital settings?
Does the contribution of PVL in different species vary?
Is PVL a factor in CA-MRSA?
How does PVL cause inflammation?
How does PVL make a big immune response and recruit so many leukocytes?
Does MRSA have other virulent factors?
Some of the mechanism is how does CA-MRSA spread?
How does Staph acquire resistance from other bacteria?
What are antibiotics or synthetic antibioitics to combat the bacteria?
Is PVL a contributer to the spread and severity of CA-MRSA?
How does MRSA infect healthy individuals?
How did MRSA become so prominent in community?
Vancomyocin is the only treatment for MRSA, what happens when MRSA becomes resistant to that?
VACCINIA
How does Vaccinia change protein production?
How does the virus “bump” off other viruses?
Why is the virus not harmful in humans?
What is vaccinia pathogenesis?
What are the research findings?
Is there any known inection associated with the virus?
RETINA
How will the information rely to the brain?
Some ganglion can work, but what if there is limited amount of functional ganglion neurons, how will the signal relay to the brain?
How will the retina be implanted into the eye?
Improvements on the vision that it can give
In producing this, how will the intpretation of color be? Will it will be black and white?
Will other parts of the eye have to be adjusted, such as the lens?
Sunday, February 21, 2010
#7 LIst of Research Questions/Problems
Posted by Unknown at 6:53 PM
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